Wiki

Losartan

Losartan (Cozaar)

Clinical Summary

Losartan is an angiotensin II receptor blocker (ARB). Documented on handwritten prescription as "Lo" — likely abbreviation for losartan (brand name: Cozaar).

Newly documented from handwritten prescription dated 2026-05-04. Dose: 50 mg once daily (afternoon, 0-1-0 dosing).

Dosing History

Date Dose Frequency Route Context
2026-05-04 50 mg Once daily (afternoon, 0-1-0) Oral Documented from handwritten Rx ("Lo")
**Major Documentation Gap**

This medication was NOT previously documented in any ingested raw source. No blood pressure readings have been ingested. Indication is PRESUMED to be hypertension and/or diabetic nephropathy protection (renoprotection in diabetes).

Indication

Presumed indications:
1. Hypertension (no BP readings documented yet)
2. Diabetic nephropathy protection — ARBs are first-line agents in diabetic patients for renoprotection, even in normotensive patients with microalbuminuria

Given Ishamma's Diabetes Mellitus diagnosis, losartan may be prescribed primarily for renoprotection rather than BP control.

Pharmacology

  • Drug class: Angiotensin II receptor blocker (ARB)
  • Mechanism: Blocks AT1 receptors, preventing angiotensin II-mediated vasoconstriction and aldosterone secretion. Results: reduced BP, reduced proteinuria, renoprotection.
  • Half-life: ~2 hours (parent drug), ~6–9 hours (active metabolite E-3174)
  • Standard dose: 25–100 mg once daily (current: 50 mg, mid-range)

Advantages in Diabetes + AML Setting

  • Renoprotection: Slows progression of diabetic nephropathy
  • Reduces proteinuria: Even in normotensive diabetic patients
  • Cardioprotection: Reduces cardiovascular events in diabetes
  • Well-tolerated: Lower cough risk compared to ACE inhibitors

Monitoring — CRITICAL

Parameter Frequency Rationale Ishamma Status
Blood pressure Weekly initially, then monthly Primary efficacy endpoint NOT YET DOCUMENTED — gap
Serum potassium Baseline, 1 week, then periodic ARBs reduce aldosterone → hyperkalemia risk K+ consistently normal (4.0 mmol/L May 4)
Renal function (creatinine, eGFR) Baseline, 1 week, then periodic ARBs can cause transient Cr rise (acceptable if <30%); contraindicated if bilateral RAS Creatinine stable 0.7–0.9 mg/dL (normal)
Microalbuminuria / UACR Baseline, then every 3–6 months Monitor renoprotection efficacy NOT YET DOCUMENTED — gap
**Hyperkalemia Risk — Monitor Potassium**

ARBs reduce aldosterone secretion, causing potassium retention. Risk factors for hyperkalemia:
- Renal impairment (Ishamma's renal function currently normal)
- Concurrent K+-sparing diuretics, NSAIDs, K+ supplements
- AML treatment effects on renal function

Current status: K+ 4.0 mmol/L (May 4) — normal. Continue monitoring.

[!info] Blood Pressure Monitoring Gap
No BP readings have been documented in any ingested lab/clinic note. Home BP monitoring or clinic BP records should be obtained to confirm hypertension diagnosis and treatment efficacy.

Drug Interactions

  • NSAIDs: Reduce antihypertensive effect, increase renal impairment risk
  • Potassium supplements / K+-sparing diuretics: Increased hyperkalemia risk
  • Lithium: Increased lithium levels (not applicable here)
  • No CYP3A4 interaction with posaconazole/venetoclax

Adverse Effects

  • Hyperkalemia (most important in renal impairment)
  • Dizziness, orthostatic hypotension (especially first dose)
  • Acute renal failure (rare, usually in bilateral renal artery stenosis)
  • Angioedema (rare, lower risk than ACE inhibitors)
  • Does NOT cause cough (unlike ACE inhibitors)

Contraindications

  • Pregnancy (teratogenic — ARBs are contraindicated in pregnancy)
  • Bilateral renal artery stenosis
  • History of angioedema with ARBs

Related

  • Diabetes Mellitus — Renoprotection indication
  • Nifedipine — Co-administered antihypertensive (calcium channel blocker)
  • Potassium — Monitor for hyperkalemia
  • Creatinine — Monitor renal function
  • Microalbuminuria — Should be monitored (not yet documented)

Medication page created during ingest of 2026-05-04 handwritten prescription.